Roger Ebert, one of America's best known film critics, has not been able to speak since he had emergency cancer surgery on July 1 to repair a burst blood vessel near the site of his June 16 surgery to remove a cancerous growth on his salivary gland. But he can still give a thumbs up or thumbs down as he reviews movies on his Web site.

Starting today, clips from Ebert & Roeper and Ebert's prior episodes of his movie review TV show will be posted here. It just may be the largest collection ever of video-based online movie reviews -- 5,000 movie reviews spanning the past 20 years will be featured.

Ebert is not quite sure when he might return to television as he awaits another surgery to restore his voice. In the meantime, he screens as many as three films a day. He watches DVDs at night to catch up on those he missed, and he's writing more than ever, he says.

The pathology results are in -- the five small polyps removed from President Bush's colon on Saturday are not cancerous.

White House Press Secretary Tony Snow announced today that microscopic evaluations of the five polyps confirm the preliminary diagnosis: tubular adenoma -- or as Snow called them: "garden variety polyps."

Nearly two-thirds of all polyps are adenomas, the vast majority of them tubular adenomas. It is not uncommon to find these polyps in routine colonoscopies.

There's nothing like a little benign news to start the day, nothing like a voice on the other end of the phone saying, "the pathology on your biopsy came back and everything is benign. We'll see you in one year for your next appointment."

While my dermatologist was freezing the pre-cancerous actinic keratoses lesions on my nose last week, she decided to cut out a suspicious chunk of skin on my hand. It was much worse than the freezing. She gave me a shot and numbed the area and then literally dug a hole into the skin just below the pinkie finger on my right hand. For days now, I've been applying antibiotic ointment, bandaging the wound, and whining about the twinges of pain that shoot through my hand.

My hand is still sore today. But I don't have skin cancer. And that makes the pain a whole lot more tolerable.

The Utah Jazz's Derek Fisher finds himself in a whole new ballgame lately as he helps his 10-month-old daughter, Tatum, fight for her life.

Tatum was diagnosed last week with retinoblastoma, a cancerous tumor in her left eye. Fisher, who was excused from his team to begin dealing with his daughter's illness, flew his family -- his wife Candace, Tatum, and Tatum's twin brother Drew -- to New York on Monday to see a specialist.

Fisher and his wife must decide on a course of treatment for their daughter. Their options are removal of the eye or a combination of surgery and chemotherapy. Their most pressing goal is to save Tatum's life. They also want to save her eye. And they think in her case, she should be able to keep her eye.

It's been a rough road for former Boise State basketball player Coby Karl, son of Denver Nuggets coach George Karl. The younger Karl, 23, had his thyroid removed 13 months ago due to papillary carcinoma, a treatable form of cancer. And he returned to the operating room just last week for the removal of cancerous lymph nodes.

Karl's recent surgery, intended to take two to three hours, lasted for seven hours. This worried Dad.

''When it goes longer and longer, you always think the worst, and start worrying about things like being under anesthesia that long and all the nightmares you have about surgeries,'' George Karl said.

But it turns out Coby was just fine -- doctors just wanted to be thorough -- and the ambitious young man plans to play ball as soon as possible.

Coby is expected to remain inactive for 10 days to two weeks. But after that, he is free to do whatever he wants. And what the 6-foot-4 guard wants to do is start working out in hopes of getting drafted by an NBA team.

''He is anxious to get into the NBA thing and celebrate basketball by trying to make it in this league,'' his dad says.

Denver Nuggets coach George Karl missed his team's match-up against the Lakers Tuesday night so he could spend some quality time with his son, Coby, who had surgery for the removal of cancerous lymph nodes on Monday.

Coby Karl, 23, spent seven hours in surgery. It was his second surgery in 13 months -- he had his thyroid removed last year after he was diagnosed with a treatable form of cancer called papillary carcinoma. Chemotherapy followed the first surgery to kill off any remaining cancer cells.

All reports indicate both Karls are doing fine and Coach Karl, who has been surviving prostate cancer since 2005, was back to his coaching duties last night. His team took on the Sacramento Kings -- and won.

Coby Karl is taking it easy for now. He just recently finished his final season with Boise State University where he led the Broncos with 14.8 points per game. He's still a great athlete, but right now, his health comes first.

"He's a young kid, he's a promising basketball player," Lakers coach Phil Jackson said. "The most important thing is his health."

White House Press Secretary Tony Snow, who on Friday asked the public not to jump to conclusions about a suspicious growth found on his abdomen, says the growth -- removed during surgery yesterday -- has been identified as cancer.

Snow, 51, told White House officials that his cancer -- first found in his colon and treated in 2005 -- has spread to his liver. He is currently consulting with doctors about chemotherapy and is reported to be feeling well.

"I'm gonna beat it again," says Snow who gave the White House deputy Press Secretary instructions for reporters.

"Tell them not to bug me," he said.

It is not clear when or if Snow, married with three young children, will return to his duties.

Boise State basketball player Coby Karl, son of Denver Nuggets coach George Karl, had surgery 13 months ago to remove his thyroid after he was diagnosed with papillary carcinoma, a form of treatable cancer. And while Karl received chemotherapy to kill off any lingering cancer cells, he must undergo cancer surgery once again.

Karl, who plans to play in the NABC All-Star game in Atlanta on March 31, will return to Boise on April 2 for surgery to remove cancerous lymph nodes.

The lymph node cancer was identified in January, but Karl, 23, kept his condition private until his team lost to New Mexico State in the Western Athletic Conference tournament semifinals. This ended the Broncos' season. And now begins Karl's second go-round with cancer.

You may remember Karl as one of last year's NBA draft hopefuls. He worked out for three teams, including his dad's team, but eventually withdrew his name from the draft and returned to Boise State for his senior year.

A little bit of education goes a long way, especially in the war against cancer. Armed with facts and figures and know-how, we can help advance prevention and early detection of this deadly disease.

So in the spirit of National Colorectal Cancer Awareness Month, here are seven truths that serve to broaden your horizons about the third most common cancer found in men and women in this country.

As you read these truths, be aware that the death rate from colorectal cancer has been on a downward climb for the past 15 years due to better screening, fewer diagnosed cases, early detection, and more advanced treatment. Keep in mind that you can help keep this trend going by raising your own awareness and by taking action on behalf of yourself and your loved ones.

• Colorectal cancer refers to cancer that starts in the colon or rectum. These cancers begin in the digestive system where food is processed to create energy and rid the body of solid waste matter.

• Colorectal cancers develop slowly over a period of years and mostly begin in the form of polyps -- growths of tissue that start in the lining and grow into the center of the colon or rectum. Removing polyps early may prevent them from becoming cancerous. More than 95 percent of colon and rectal cancers are called adenocarcinomas.

• For people of average risk, screening is recommended beginning at age 50. Those whose risk is higher than average should talk with a physician about appropriate screening.

• Screening is used to detect disease in people who do not have any symptoms. In many cases, screening tests find colorectal cancers at an early stage and greatly improve the chances of successful treatment. Screening tests can prevent some cancers by allowing doctors to find and remove polyps that might become cancer. There are several tests used to look for colorectal cancer. Ask your doctor what test is best for you.

• Treatment for colorectal cancer includes surgery, radiation therapy, chemotherapy, and newer targeted therapies.

• The American Cancer Society predicts there will be 112,340 new cases of colon cancer and 41,420 new cases of rectal cancer in 2007 in the United States. Combined, the diseases will cause about 52,180 deaths.

• The Colorectal Cancer Coalition -- or C3 -- is a national organization whose mission is to eliminate suffering and death due to colon and rectal cancer through advocacy. Visit here for more information.

The material shared in this post was gathered from the websites of the American Cancer Society and the Colorectal Cancer Coalition.

A friend of a friend was diagnosed this week with a cancerous brain tumor -- a glioma to be exact -- and the surgery to remove the mass is scheduled for Monday.

I don't know much about this woman or her cancer, but I do know doctors told her yesterday she will likely survive for only a few years. I can't help but think that if doctors had given me this same prediction at the time of my cancer diagnosis, my time would just about be up.

I can't fully grasp the magnitude of this sad and sobering news. But I can comprehend that any one of us could be on the receiving end of such an announcement at any given moment. We are all vulnerable. And so I am confronted once again with the powerful and painful reminder that each day really could be my last.

Researchers have found that when cells become cancerous, they become 100 times more likely to genetically mutate than non-cancerous cells. This explains why tumor cells have so many mutations. Good news on the research front. But bad news on the treatment front -- because therapies that target a certain gene may be largely ineffective in controlling cancer.

"This is very bad news, because it means that cancer cells in a tumor will have mutations that protect them from therapeutics," says lead researcher Dr. Lawrence Loeb, professor of pathology and biochemistry at the University of Washington School of Medicine in Seattle, who presented his findings February 18 at the meeting of the American Association for the Advancement of Science in San Francisco.

Loeb says chemotherapy drugs target specific oncogenes -- genes that affect the malignancy of a cell -- but if cancer cells are mutator cells, then a single tumor may have cells with all sorts of oncogenes. And while chemotherapy may kill some cancerous cells, millions of others will live on.

It's not all bad news, though, says Loeb who believes this research may help doctors determine the stage and malignancy of tumors by testing the number of mutations. It may also help researchers understand what makes a cancer cell a mutator and how to slow the rate of mutation.

"The idea is that if you might normally get exposed to something in the environment at 20 years old that would give you cancer by age 55, then if we cut the mutation rate in half, you might not get cancer until age 90, and you may even die of something else before that," Loeb explained.

Katherine Schaefer was investigating methods for treating the inflammation seen in Crohn's disease and ulcerative colitis when something terrible happened -- she noticed her carefully cultured cells were dead. And then something wonderful happened -- she realized she had stumbled upon a potential new method of attacking cancerous tumors that have become resistant to existing drugs.

Schaefer and her colleagues at the University of Rochester Medical Center in New York were testing a compound called a PPAR-gamma modulator -- a compound never considered a cancer drug, or a drug of any kind really -- when Schaefer made a calculation error and used a lot more of the compound than she should have. And her cells died.

Upon further study, Schaefer found the compound killed just about every possible epithelial tumor cell. These cells line organs such as the colon and also the skin. The compound, that works like taxane drugs but without eventual tumor resistance, also killed colon tumors in mice without making them sick.

The research team, whose findings are published in the journal International Cancer Research, plans more safety tests in mice. And eventually, if their outcomes are promising, they plan to design something they can patent as a new drug -- because they would love to see this disastrous lab experiment one day lead to treatment for cancers of the colon, esophagus, liver, and skin.

In the January issue of the Journal of the National Cancer Institute, findings were published that says calcium seems to protect high-risk people from developing polyps in the colon. Polyps are growths in the colon and some can become cancerous over time.

Patients, who had a history of benign polyps, either took 1,200 milligrams of calcium in supplement form or received a placebo daily for four years. In this study it showed that calcium use was associated with a 17 percent lower risk for polyp recurrence.

"It really does look like calcium interferes with carcinogenesis in the large bowel" researcher John A. Baron, MD, tells WebMD. "The fact that this reduction in risk persisted for years after people stopped taking calcium is amazing".

Baron also stated that it is still not clear if the benefits outweigh the risk since some studies have linked calcium treatment with an increase in prostate cancer risk.

On the flip side a University of Arizona epidemiology professor Maria Elena, PhD, worries that the findings will make people think that all they need to do to protect themselves from colorectal cancer is to increase their calcium intake, which in men high doses can be dangerous.

The best way to prevent colon cancer is to get a colonoscopy when you turn fifty and make sure you have follow-ups. That is not as easy as popping a pill but its true.

The American Cancer Society recommends that adults aged 19 to 50 take in 1,000 milligrams per day of calcium and that those over 50 get 1,200 milligrams. The guidelines stress that the calcium should come primarily from food sources and not supplements.

One or more extra or missing chromosomes can both fuel tumor growth and act to suppress tumor growth, according to University of California, San Diego (UCSD) School of Medicine researchers.

Heralded as a discovery that solves a 100-year-old genetic puzzle because the hypothesis was first suggested by German biologist Theodor Bover that long ago, researchers sought to determine if the wrong number of chromosomes contributed to tumor growth, or was a consequences of damage in cancerous cells.

While studying aneuploidy -- which is what the occurrence of one or more extra or missing chromosomes is called -- in mouse models, the researchers found that the same genetic mechanism that promotes tumor growth can slow tumor growth.

"This study opens up a whole series of potential therapeutic targets for cancer," said Beth A.A. Weaver, of the Ludwig Institute for Cancer Research and UCSD Department of Cellular and Molecular Medicine, the study's first author. "By increasing the level of genetic damage, we can kill tumor cells."

A colonoscopy camera lets the physician check for abnormalities inside the colon. These can include cancerous or precancerous growths. The doctor guides a flexible scope though the colon, that can take about seven minutes, he then spends on average another six minutes withdrawing the scope evaluating inside of the colon.

The New England Journal of Medicine published a study that found colonoscopies that took a longer time to complete found more abnormal growths. Faster testing was shown to miss some abnormalities. Doctors who spent more than six minutes withdrawing the colonoscopy tube found more abnormal growths than those who withdrew it in less than six minutes.

The study did not have a conclusive answer as to exactly long physicians should spend withdrawing the tube. Other experts say to keep it in the range of six to ten minutes.

I know this is the last thing you want to say to your physician-- "Can you keep that up there a bit longer please?', but it might just save your life.