Who knows which pieces of cancer information floating around out there are actually true? I don't. Do cell phones cause cancer? Some say yes, some say no, I say I'm confused! Luckily, I happened upon this Discovery Health article that highlights a variety of myths and then offers the lowdown on each one. Here are seven of them:

Myth #1. There is currently a cure for cancer, but the medical industry won't tell the public about it because they make too much money treating cancer patients.

Chalk this up to urban legend. And consider this: doctors, laboratory scientists, and their families and friends die of cancer at the same rate as everyone else in the United States. How about this: medical breakthroughs happen all the time and are quickly applied. Think about antibiotics and vaccines -- like the polio vaccine -- that have transformed health care. How about this? Not too long ago, less than one in 10 kids with leukemia survived 10 years. Now, the cure rate is nearly 80 percent. Seems like progress to me.

Yesterday I visited the High Points Monument at High Point State Park. It was a fitting day to visit the monument -- Memorial Day. The monument was built and dedicated to the memory of New Jersey's wartime heros. Construction was started in 1928 and completed in 1930.

My husband and I climbed the stairs up the 220- foot structure for a breathtaking view of the ridges of the Pocono Mountains, the Catskill Mountains and the Wallkill River Valley.

The high points in my life can come unexpectedly. I think we should all think about what the high points in our lives have been and cherish the memories. A camping trip in an RV, my husband and two dogs this weekend was definitely a high point in my life. High points don't have to be something monumental -- no pun intended. High points can be small things that make the day a joyful one.

Find as many high points in your life as you can.

Living With Cancer is an inspirational documentary that will air on the Discovery channel tonight at 8pm EDT. Hosted by Ted Koppel, it chronicles the experiences of Leroy Sievers, an executive producer at Discovery.

In 2002 Sievers was diagnosed with colorectal cancer that had metastasized to his lungs and brain. The program features a discussion with Sievers as he deals with chemotherapy, scans and cutting edge procedures.

Koppel says the program is intended to show that people can and do live successfully with cancer everyday.

Sievers says "Normalcy is the greatest gift anyone can give you, let me continue to be me, not cancer. I am not my disease, I just have a disease. That's all"

Tune in -- I know I will.

The way I see it, there are two threads common to the journeys of many cancer patients and survivors -- the desire to hear stories of others walking in similar shoes and the desire to be heard.

These threads have definitely woven themselves throughout my own cancer experience. When first diagnosed, I searched long and hard for personal stories of women struggling with and conquering breast cancer. Once I was well on my road to recovery, I wanted others to hear my story, in hopes it would resonate with those looking for the same comfort I once craved.

Won't you consider sharing your story too? It might just help you. It will surely help others.

Think about this:

On the Discovery Health website, an offer awaits you.

"We would love to hear your story," reads a portion of the site. "So would the many others who share your relationship to Cancer."

If you are at least 18 years of age and have a few minutes to spare, simply call 888-890-6339 and follow the instructions to record your voice. When all stories are ready to be published in podcast format, an online notice will be posted.

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Researchers have made a stem cell discovery that may help treat pancreatic cancer -- one of the deadliest forms of the disease.

University of Michigan scientists have found cancer stem cells in pancreatic tumors that appear to drive cell tumor growth and could lead to the development of drugs to target and kill these cells.

Pancreatic cancer kills 97 percent of people diagnosed with the disease within five years. Half of all diagnosed patients die within six months of diagnosis, and this cancer -- that spreads quickly and is rarely detected at an early state -- kills 33,000 each year in the United States alone. So any improvement in the study of this disease is a true gift.

"The clinical implications of this work are significant," said Dr. Diane Simeone, director of the Gastrointestinal Oncology Program at the University of Michigan Comprehensive Cancer Centre and lead author of the study, published in the journal Cancer Research.

"We've made baby steps in improving the survival in these patients -- on the order of a few months (longer to live) -- over the past decade or so. But we really haven't had a major breakthrough in coming up with something that has the potential to provide a cure," she said.

Simeone says killing these cancer stem cells is like pulling out the root of a weed. And she says the best way to pull out the root is to target these stem cells instead of the traditional approach of shrinking tumors by killing as many cells as possible -- an approach that may be flawed because cancer stem cells tend to resist standard therapies.

Canadian researchers have made an unexpected discovery in a molecule that appears to drastically boost the ability of standard drugs to kill breast cancer cells. Currently, the discovery has been confined to the lab -- but researchers hope the power of this molecule, the ANK peptide, can one day be used to counter drug resistance for many women with breast cancer.

Scientists from Queen's University say the ANK peptide, not a drug by itself, gives drugs like taxol and nocodazole more than triple the ability to kill breast cancer cells. One scientist says the process of enhancing drug effectiveness is much like adding flavor to coffee to make it taste better.

This is exciting news, but the results -- published Monday in the journal Cancer Research -- only apply to lab experiments at this point. Researchers must now proceed with testing the peptide-drug combination in lab mice. If successful, they will move on to human testing. The whole process could take years. But early results are so promising that application for a U.S. patent on the peptide has already been made.

According the medical experts, breast cancer patients can become resistant to some drugs depending on duration of treatment, dose of medication, and genetic makeup.

"This peptide would be able to give them another chance," said a researcher from this study. "For those who respond reasonably well, they will do even better; for those who don't respond to this drug treatment ... we greatly hope this will make the current drug more useful by extending its impact to a wider range of people, particularly those with a resistance problem."

One or more extra or missing chromosomes can both fuel tumor growth and act to suppress tumor growth, according to University of California, San Diego (UCSD) School of Medicine researchers.

Heralded as a discovery that solves a 100-year-old genetic puzzle because the hypothesis was first suggested by German biologist Theodor Bover that long ago, researchers sought to determine if the wrong number of chromosomes contributed to tumor growth, or was a consequences of damage in cancerous cells.

While studying aneuploidy -- which is what the occurrence of one or more extra or missing chromosomes is called -- in mouse models, the researchers found that the same genetic mechanism that promotes tumor growth can slow tumor growth.

"This study opens up a whole series of potential therapeutic targets for cancer," said Beth A.A. Weaver, of the Ludwig Institute for Cancer Research and UCSD Department of Cellular and Molecular Medicine, the study's first author. "By increasing the level of genetic damage, we can kill tumor cells."

Deep within the pages of ancient texts detailing the remedies used by Chinese medicine practitioners, is there a cure for cancer waiting to be rediscovered? The global pharmaceutical company Merck thinks there might be a reference or two to natural cancer-fighting products used by healers then that is obscurely hidden and not known now in modern western medicine.

Merck has entered into a deal with Hong Kong's Chi-Med to look for evidence of promising products that the pharmaceutical company can research and test in clinical trials. According to the article Merck looks for ancient Chinese cancer cure written by Susie Mesure, "Western pharmaceutical companies are increasingly outsourcing their drug discovery work, with many looking east for the solution to medical mysteries that Western doctors cannot solve."

Traditional Chinese Medicine, TCM, is a practice of medicine that combines medicinal herbs, nutrition, meditation, massage, exercise and acupuncture with an applied philosophy in the harmonious balance of yin and yang for treating illness. In all fairness, because this system of medicine has developed over thousands of years, and my understanding limited by Western educational influence, the definition I have given is a very brief, and possibly incomplete, overview of TCM. If you are interested in learning more about TCM, begin by visiting Traditional Chinese Medicine at Wikipedia.

Chi-Med will be scanning information in a library of 10,000 natural substances for those that might hold potential in a cure for cancer. It will be interesting what they find.

In a surprising discovery, 200 mutated genes linked to the development, growth and spread of breast and colon cancers have been identified by Johns Hopkins Kimmel Cancer Center scientists. This information might provide vital research into the future treatment for these cancers.

Other cancers can be studied using the methods these researchers, called the Hopkins gene hunters, used in discovering the 200 genetic mutations for breast and colon cancers.

"This gives us some understanding of why breast and colon cancers, and most likely other cancers as well, are very different diseases and develop through different processes. When we say this will drive cancer research for the next couple of decades, this is one of the reasons," states Kenneth Kinzler, Ph.D., professor of oncology and co-director of the Ludwig Center at Johns Hopkins. "Now researchers will study how these mutations occur in breast and colon cancers, perhaps searching for environmental agents or cellular processes that drive these changes."

To read more detail on this study and discovery, visit Genome Code Cracked for Breast and Colon Cancers. An MP3 file and XML for iPods has been provided so that you can listen to Johns Hopkins Kimmel Cancer Center scientists discuss this research.

I really do believe deep down in my gut that I will survive breast cancer -- that I will witness the wonder of my children growing up, that I will be married long enough that the years blur together, that I will live to a ripe old age. But I still have moments of doubt -- moments powerful enough to make me think I should not have a third child, just in case cancer comes back. To combat these moments -- that seem to surface more now that my treatment has stopped -- I try to keep busy, keep my mind occupied, keep living. My steps for surviving in the short-term include writing, journaling, exercising, relaxing, and spending time with family. But I also follow some steps for long-term survival -- steps that transcend the moment and give me purpose and direction. And here are seven of them.

I love trees. They are mighty and mysterious. It's no wonder that we humans have found out that they contain a cancer-killing agent in their bark extract. The pacific yew tree, found in the Pacific Northwest coastal region of the United States, was found to have bark extract that tested positive for cytotoxicity (substance poisonous to cells). This discovery in the early sixties has led us to what we know today as Taxol and Taxotere.

Taxol comes from the pacific yew tree (Taxus brevifolia) and Taxotere comes from the European pacific yew tree (Taxus baccata). These drugs treat a variety of different cancers including breast, ovarian and lung cancers.

The pacific yew tree was considered worthless as lumber. Botanist Arthur S. Barclay, PHD, sent a specimen of bark to chemist Monroe Elliot Wall, PHD. The testing of the bark extract led to a drug that interferes with the development of cell duplication. This was a different way to attack a cancer cell. Some other chemotherapy treatments work to interfere with a cancer cell's DNA.

The Taxenes gave us a new angle to combat the fast growing cancer cells. Of course, most chemotherapy agents do that at a price. These drugs do have side effects since they can't tell the difference between fast growing cancer cells and other fast growing cells in our body.

What other untapped resources are out there in our forest and in nature that could help save lives? We already know of organisms in China, Japan, New Zealand, Africa, Madagascar and the Caribbean Sea that have yielded drugs to treat cancer.

This isn't exactly natural therapy but it feels much more natural when the treatment derives from tree bark.

George W. Bush declared five years ago that no federal funding would be allocated for embryonic stem cell research. He has not changed his mind -- and two weeks ago vetoed a bill that would allow this research. He says that supporting the bill would be supporting the taking of innocent human life in order to find medical benefits for others. For Bush, it crosses a moral boundary. But most Americans support stem cell research and would like to see the Bush White House loosen its restrictions, says Kathy Hudson, director of the Genetics and Public Policy Center at Johns Hopkins University in Washington. Until this happens, though, scientists and researchers find themselves in an ethically-charged minefield, operating carefully and responsibly and ethically.

Dr. Peter Dirks -- a neurosurgeon specializing in childhood brain cancer at Toronto's Hospital for Sick Children who has been making headlines around the world for isolating brain cancer stem cells two years ago -- is one person weighing in on controversial issue of stem cell research. Dirks does not use embryonic stem cells but instead uses cells from tumors removed during brain surgery that would otherwise be discarded. But he says embryonic stem cell research is critical for his success. It's what has led to the findings that exist today -- and it holds the clues for further discovery. It's a delicate matter, though, regardless of the politics surrounding the issue. Before he harvests stem cells from any patient's brain tumor, for example, Dirks asks parents to sign a three and a half-page consent form. And that's just the beginning.

Federally-funded embryonic stem cell research does not yet have its beginning here in the United States. Maybe the tides will change. Maybe we will see progress. Maybe we won't. Only time will tell.

Eccentric hotelier and television personality Tim Hadcock-Mackay, who is reported to have been an active fundraiser for Sargent Cancer Care for Children, was found hanged. While the police investigating do not suspect foul play, nothing at this time has been ruled out.

According to news reports, Hadcock-Mackay was found in the woodlands behind his lavish 19th century manor Barnby Moor Hall. At the time of his death, Hadcock-Mackay and life partner Torquil Mackenzie-Buist were in talks for a new television show featuring their home to the public called Mayhem At The Manor.

"The normal response of prostate cells when male hormones are blocked is cell death," said George Kulik, assistant professor of cancer biology and senior researcher at Wake Forest University School of Medicine. "The cancer cells find a way to resist the treatment and we wanted to discover the mechanism."

Researchers have discovered the canny and shrewd way prostate cancer cells manage to defy death and become resistant to hormone treatment. The prostate cancer cells are using three separate pathways to send signals to cancer cells by inactivating a protein, BAD, meant to cause cell death.

If animal and human studies further prove what the researchers have discovered, new drug therapies could be developed to keep BAD active and doing its job in helping to destroy prostate cancer cells.

The topic of my hair is often the subject of conversation -- and is a constant reminder that this brown curly hair I have covering my head is nothing like the straight blond hair I was born with, grew up with, was known for. Because my little boys have white blond hair, I am consistently asked by strangers, "Where did your boys get that blond hair?" "From me," is what I want to say because it's the truth -- but that would make no sense to anyone who does not know me, anyone who does not know that my hair -- that once looked much like my boys' hair -- was lost to chemotherapy and returned shockingly different. So sometimes I just chuckle in wonder with these strangers who may not expect an answer anyway. Or I tell them the story -- if they seem to really want in on the details of the mystery. Most people are surprised that my hair grew back like it did. I am not surprised -- I was warned that it might happen -- although it is still a startling discovery each time I look in the mirror, each time I look back at photos, each time I see gray hairs emerging through my dark hair -- gray that only slightly showed up in the midst of my blond locks.

The memory of my blond hair keeps popping up. My husband told me the other day that he had a dream about me -- I was in a restaurant, at a table, by myself. He was walking toward me. And I had blond hair. The rest of the dream is insignificant. The blond hair is significant. And the other day, I pulled my brush out of my purse. It hasn't been used in more than a year -- because I don't brush my curls at all -- and at the base of the brush, wound around the bristles, were long blond strands of hair. My blond hair. My old hair. The same hair I showed my friend who visited from Ohio last week -- the hair that was once on my head, was cut off in preparation of the great fallout, and is now kept in a ziplock bag.

I like my brown hair. I like my curls. But I miss my blond hair. I am sad that I no longer match my children, that I don't look like the bride in my wedding photo, that I will attend my 20-year high school reunion in two years and will wear a photo name tag that looks nothing like me. I like the familiar -- which is why I never wanted to show my bald head, why I covered my head with blond wigs and hats to keep my appearance as close to normal as possible. And then in a strange turn of events, my hair grew back in an unfamiliar fashion -- and somehow the question, "I see where your boys get that blond hair" flip-flopped into "Where did your boys get that blond hair?" It is all still new to me. I know one day it will become familiar and normal and not such a big deal. Some day. I hope.