It might not be possible at this time to eradicate cancer altogether. But we may be able to stop cancer cells in their tracks through a process called senescence. In senescence, cells don't divide. And when cells don't divide, they don't grow. In such a scenario then, cancer cells wouldn't divide and therefore couldn't grow.
Think about this:
According to lab tests on mice, triggering senescence in certain cells hampers the growth of some tumors.
Researchers at the University of Texas M.D. Anderson Cancer Center in Houston are the ones behind the scenes on this project -- the study appears online in EMBO Reports, a publication of the European Molecular Biology Organization -- and all eyes are on the p53 gene.
The p53 gene lives within cells and works to nip cancer in the bud by springing to action in damaged cells that may be spinning themselves into a cancer frenzy.
Researchers say senescence ordered by the p53 gene is extremely important in suppressing tumor formation and is as important as apoptosis -- a type of programmed cell death. But in some cancers, senescence might not be enough to halt cancer, they found.
OK, so senescence is not a perfect approach to halting all cancers. But it seems to work for some -- so I say for now, let's take what we can get.
Cancer is not an inevitable outcome in the process of aging, but the older a person gets, the greater the odds for developing cancer. Aging have long been known as one of the greatest risks to developing cancer -- and not one that anyone has ever been able to alter. 
One of the most important factors in finding a cure for cancer is to figure out what causes our cells to change and become malignant. A study was done by Edinburgh scientists at the University of Edinburgh Cancer Research Centre.
